Gender medicine
Gender Scientific Societies
• Società Italiana per la Salute e la Medicina di Genere
Italian Society of Gender Health and Medicine
• International Society of Gender Medicine
• Home
• Research Centers
• Berlin Institute of Gender in Medicine
• Brigham and Women’s Hospital
http://cgws.upm.edu.ph/news-jan-june07.html
• Center for Gender Based Biology – UCLA
http://www.genetics.ucla.edu/gendercenter/
• Center for Gender and Women Studies – University of Philippines Manila
http://cgws.upm.edu.ph/news-jan-june07.html
• Cleveland Clinic
http://my.clevelandclinic.org/heart/disorders/heartfailure/hfwomen/default.aspx
• Gender and Medicine Research Unit – Monash University
http://www.mihsr.monash.org/centres/gendermed.html
• Karolinska Institutet
http://ki.se/ki/jsp/polopoly.jsp;jsessionid=aQs6fxfKghne02Csj0?l=en&d=8993
• Jewish Hospital & St. Mary’s Health Care
http://www.jhsmh.org/carecenters/oc_med_women.html
• Nigerian Center for Gender, Health and Human Rights
http://ncghhr.tripod.com/humanrights/
• Women’s Health – Stanford
http://womenshealth.stanford.edu/research/
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• Clinical Departments
• Azienda Ospedaliero-Universitaria, Policlinico di Modena
http://www.policlinico.mo.it/dipartimenti/dipartimento8.asp
• Center for Cardiovascular Research, Charitè Berlin
http://www.ccr.charite.de/site/html/en/ag_regitz.html
• Department of Women and Gender in Medicine – Institute of Public Health, University of Copenhagen
http://www.pubhealth.ku.dk/mkk_en/
Master in UK
Exeter University
MA in Sexuality and Gender Studies
MRes in Sexuality and Gender Studies
Institute of Education
MA in Education, Gender and International Development
London Metropolitan University
Gender Studies MRes
Modern British Women's History MA by Research
London School of Economics and Political Science
MSc Gender / MSc Gender (Research)
MSc Gender and Social Policy
MSc Gender and the Media
MSc Gender, Development and Globalisation
Queen’s University Belfast
MA Gender and Society
University of Birmingham
MPhil(B) Gender Studies
University of Edinburgh
MSc Gender History
University of Leeds
MA Gender Studies
MA Gender Studies & History & Philosophy of Science
MA Gender Studies (Research)
MA Gender, Management & Organisation
University of Manchester
Diploma in Gender, Sexuality and Culture
MA in Gender, Sexuality and Culture
University of York
MA Women's Studies
MA Women's Studies (Humanities)
MA Women's Studies (Social Research)
MA Women's Studies by Research
MA Women, Violence and Conflict
University of Warwick
MA in Gender and International Development
MA in Gender, Literature and Modernity
MA in Interdisciplinary Gender Studies
MA in Social Research with Specialism in Interdisciplinary Gender Studies
Master in Spain
Universitat Autonoma de Barcelona
http://www.uab.es/servlet/Satellite?cid=1096481120166&pagename=UAB%2FPage%2FTemplatePageDetallEstudisPOP¶m1=1096482864242¶m2=&setletertype=_aa
Master in Germany
University of Berlin
http://www.charite.de/health-society/
Gender dimorphism in adipose tissue response to stress conditions: A plausible mechanism to explain the conflicting data regarding trauma and obesity. 2016
INTRODUCTION Obesity is a chronic low grade inflammatory condition associated with the elaboration of proinflammatory cytokines and adipokines from adipose tissue. Gender dimorphism, (in part due to sex hormones) has been identified after injury and hemorrhagic shock. We hypothesized that the sex hormones estrogen (E2) and testosterone (DHT) have disparate effects on inflammatory mediator production from adipose tissue under stress conditions. This was studied in an in vitro model.
METHODS Mature adipocytes differentiated from adipose-derived stem cells (ADSC) were co cultured (2:1) with macrophages (RAW 264.7) and subjected to hypoxia-reoxygenation (H/R), and/or incubation with physiologic (10μM) or stress (10μM) concentrations of epinephrine (epi). E2 or DHT were added in a range of physiologic concentrations and culture supernatants were obtained 12 hours post incubation and TNFα, IL-6 and adiponectin levels were measured by ELISA.
RESULTS Basal TNFα and IL-6 release from co-cultures was significantly increased in response to epi and/or H/R conditions. E2 decreased cytokine release to basal levels, whereas DHT had no effect. Of note, varying the concentration of epi had no effect on cytokine release. Basal levels of adiponectin release were significantly decreased in response to epi and/or H/R conditions. E2 exposure returned adiponectin levels to basal levels, whereas DHT had no effect. The inverse of this relationship was observed in regard to the sex hormones effect on leptin release. E2 returned leptin release to basal levels whereas DHT had no effect.
CONCLUSION Stress levels of epi and H/R increased proinflammatory cytokine production and decreased adiponectin levels in adipocyte co cultures. E2 at physiologic concentrations decreased TNFα, IL-6 and preserved adiponectin levels following Epi and/or H/R conditions. There was no effect of DHT on mitigating the proinflammatory response. Our results suggest a gender dimorphism in adipose tissue under stress conditions that may explain the conflicting data in the literature.
