A short yet comprehensive description
Reference methodologies, based on ion chromatography and flame atomic emission spectrometry are equivalent, and able to satisfy current analytical specifications for serum potassium measurements.
Survey of serum potassium reference measurements. 1999
ANALYTICAL TRICKS AND TIPS
THE BIOLOGICAL CONTEXT
Hormonal and pharmacological modification of plasma potassium homeostasis. 2010
Adults: 3.5-5 mEq/L (3.5-5 mmol/L)
Premature neonates, cord blood: 5-10.2 mEq/L (5-10.2 mmol/L)
Premature neonates, 48 hours: 3-6 mEq/L (3-6 mmol/L)
Neonates, cord blood: 5.6-12 mEq/L (5.6-12 mmol/L)
Neonates: 3.7-5.9 mEq/L (3.7-5.9 mmol/L)
Infants: 4.1-5.3 mEq/L (4.1-5.3 mmol/L)
Children: 3.4-4.7 mEq/L (3.4-4.7 mmol/L)
Reduction of erythrocyte (Na(+)-K+) ATPase activities in non-insulin-dependent diabetic patients with hyperkalemia. 1992
To elucidate the mechanism of hyperkalemia in diabetic patients without renal failure, we investigated (Na(+)-K+) adenosine triphosphatase (ATPase) activity in erythrocyte membrane, erythrocyte Na+ and K+ content, and plasma endogenous digitalis-like substance in control subjects (n = 16) and non-insulin-dependent diabetes mellitus (NIDDM) patients (n = 62).
[Diabetes mellitus as a general membrane disease and its consequences]. , 2001
Untreated diabetic patients generally show hyperkalemia. Forceful treatment with insulin of these subjects often causes severe hypokalemia as a consequence of sudden translocation of the Na(+)-pump.
Specificity, sensitivity etc.
PROs and CONTROs